摘要：炎症是免疫系统的一种生物学反应，可由多种因素触发，包括病原体、受

摘要：炎症是免疫系统的一种生物学反应，可由多种因素触发，包括病原体、受损细胞和有毒化合物。这些因素可能在心脏、胰腺、肝、肾、肺、脑、肠道和生殖系统引起急性和/或慢性炎症反应，可能导致组织损伤或疾病。感染性和非感染性因子和细胞损伤都会激活炎症细胞并触发炎症信号通路，最常见的是NF-κB、MAPK和JAK-STAT通路。下面将重点介绍炎症的病因、炎症反应机制、炎症消退和器官特异性炎症反应。关键词：炎症病因；炎症机制；炎症器官

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结果 (英语) 1: [复制]

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Abstract: Inflammation is a biological response of the immune system that can be triggered by a variety of factors, including pathogens, damaged cells, and toxic compounds. These factors may cause acute and/or chronic inflammatory responses in the heart, pancreas, liver, kidney, lung, brain, gut and reproductive system, potentially leading to tissue damage or disease. Both infectious and non-infectious agents and cellular damage activate inflammatory cells and trigger inflammatory signaling pathways, most commonly NF-κB, MAPK, and JAK-STAT pathways. The following will focus on the etiology of inflammation, inflammatory response mechanisms, inflammation resolution, and organ-specific inflammatory responses. <br>Key words: inflammatory etiology; inflammatory mechanism; inflammatory organ

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结果 (英语) 2:[复制]

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Abstract: inflammation is a biological response of the immune system, which can be triggered by a variety of factors, including pathogens, damaged cells and toxic compounds. These factors may cause acute and / or chronic inflammatory reactions in the heart, pancreas, liver, kidney, lung, brain, intestine and reproductive system, which may lead to tissue injury or disease. Both infectious and non infectious factors and cell damage activate inflammatory cells and trigger inflammatory signaling pathways, the most common being NF- κ B. MAPK and JAK-STAT pathways. The following will focus on the etiology, mechanism, regression and organ specific inflammatory response of inflammation.<br>Key words: inflammatory etiology; Inflammatory mechanism; Inflammatory organ

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结果 (英语) 3:[复制]

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Abstract: Inflammation is a biological reaction of immune system, which can be triggered by many factors, including pathogens, damaged cells and toxic compounds. These factors may cause acute and/or chronic inflammatory reactions in the heart, pancreas, liver, kidney, lung, brain, intestine and reproductive system, and may lead to tissue damage or diseases. And infectious and non-infectious factors and cell damage can activate inflammatory cells and trigger inflammatory signal pathways, the most common of which are NF-κB, MAPK and JAK-STAT pathways. The following will focus on the etiology of inflammation, inflammatory response mechanism, inflammation regression and organ-specific inflammatory response. Key words: etiology of inflammation; Inflammation mechanism; Inflammatory organ

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