在大鼠中，应激反应中恐惧感的增加也与海马神经发生和突触密度的降低有关（

In rats, the increased fear in the stress response is also related to hippocampal neurogenesis and decreased synaptic density (8, 9). In addition to the changes in hippocampal GR expression, the mother’s LG-ABN behavior enhancement in the first week after birth is related to hippocampal neuron survival, synapse formation and cognitive improvement under stress (4, 10, 11) . These findings indicate that maternal care has a wide range of effects on hippocampal gene expression. Importantly, cross-culture studies provide evidence of a direct relationship between maternal love and hippocampal gene expression, stress behavior response, and hippocampal development. Therefore, the biological offspring of low LG-ABN female mice raised by high LG-ABN female mice is similar to the normal offspring of high LG-ABN female mice (and vice versa). These findings indicate that changes in maternal behavior can directly plan basic defense responses to stress and serve as a mechanism for the transmission of individual differences in stress responses across generations (3, 5, 12)

In rats, an increase in fear in stress responses was also associated with a decrease in the neurogenes of the sea mass and synapse density (8,9). In addition to changes in the expression of the sea mass GR, the increased LG-ABN behavior of the mother during the first week of life was associated with improved cognitive ability in the mhairel neuron survival, synth formation, and stress state (4,10,11). These findings suggest that maternal care has a fairly wide range of effects on the expression of the hema gene. Importantly, cross-cultivation studies provide evidence of a direct relationship between maternal love and the expression of the hema's genes, stress behavioral responses, and the development of the hema. As a result, the biological offspring of low LG-ABN female mice raised by high LG-ABN female mice are similar to the normal offspring of high LG-ABN female mice (and vice versa). These findings suggest that changes in medding can directly plan the basic defense response to stress and act as a mechanism for intergeneral transmission of individual differences in stress response (3, 5, 12)

In rats, the increase of fear in the stress response was also related to the neurogenesis of hippocampus and the decrease of synaptic density (8,9). In addition to the change of GR expression in hippocampus, the enhancement of lg-abn behavior in the first week after birth was related to the improvement of hippocampal neuronal survival rate, synaptic formation and cognitive ability under stress (4, 10, 11). These findings suggest that maternal care has a wide range of effects on gene expression in the hippocampus. This study provides important evidence for the relationship between hippocampal development, cross reaction and maternal love. Therefore, the biological offspring of low lg-abn female mice raised by high lg-abn female mice are similar to normal offspring of high lg-abn female mice (and vice versa). These findings suggest that changes in maternal behavior can directly plan the basic defense response to stress and serve as a mechanism for the transmission of individual differences in stress response across generations (3,5,12)<br>