Our study indicates that this effect cannot be considered as a primary source for stimulation of ketogenesis in our subjects because the elevated T level was stable throughout the dietary intervention. In favor of such an assumption is the fact that despite elevated T levels, the concentration of ketone bodies (β-HB) returned to baseline values after the CHO re-feeding. Human studies have demonstrated strong positive relationships between dietary saturated fat and T, which can be considered as a predictor of plasma T level