In this study, AT could significantly improve HFD-induced antioxidant dysfunction, such as increasing the expression of CAT, SOD, FRAP and HO-1 and reducing the elevation of MDA.
In this study, at can significantly improve HFD induced antioxidant dysfunction, such as increasing the expression of cat, SOD, FRAP and HO-1 and reducing the increase of MDA.
In this study, AT can obviously improve the antioxidant dysfunction induced by HFD, such as increasing the expression of CAT, SOD, FRAP and HO-1 and reducing the increase of MDA.