Legend to Figure:Main laboratory results of a 63-year old black male p的简体中文翻译

Legend to Figure:Main laboratory re

Legend to Figure:Main laboratory results of a 63-year old black male patient admitted for acute respiratorydistress associated to novel coronavirus disease (COVID-19) are shown in the upper panel(A). He rapidly developed acute kidney injury without hemodynamic compromise. Hisrespiratory status improved but inflammatory syndrome persisted and renal function furtherdeteriorated. Illustrative images of his kidney biopsy are shown in the lower panel B, C andD). Light microscopy study (FAOG stain, original magnification x 200 (B and D) and x 400(C)) showed: 1) a severe collapsing glomerulopathy (focal segmental glomerulosclerosis)characterized by the global collapse of shrinking capillary loops (B and C) and thedetachment from the basement membrane of hypertrophic, proliferating podocytes(“cobblestone pattern”, *, B) which contained numerous proteins reabsorption vacuoles (*,C). 2) acute tubular lesions (D) with focal tubular necrosis, dilatation and the presence ofintratubular reabsorption vacuoles (*, D) reflecting the heavy proteinuria.Immunofluorescence study did not show any significant immune deposits.Electron microscopy study (E, x 15000 and F, x 73000) disclosed within the podocytescytoplasm vacuoles containing numerous spherical particles (*, E) measuring between 50 to110 nm and surrounded by spikes measuring 9-10nm (*, F). These particles have the typicalappearance (“solar corona”) of viral inclusion bodies reported with the emerging severe acuterespiratory syndrome coronavirus 2 (SARS-CoV-2)3.Abbreviations: SCr, serum creatinine. UP/Cr. Urinary protein over creatinine ratio. UAlb/Cr, urinaryalbumin over creatinine ratio. CRP, C-reactive protein. SARS-CoV-2, severe acute respiratorysyndrome coronavirus 2. WBC, white blood cell count. PN, polynuclear neutrophils. Lym,lymphocytes. Hb, haemoglobin. Plt, platelet count. IL, interleukin. IFN, interferon. CCL, CXCL, TNF,tumor necrosis factor. CH50, haemolytic complement activity 50%. AP50, alternative pathwayactivity 50%. sC5b-9, soluble C5b-9. HIV, human immunodeficiency virus. CMV, cytomegalovirus.
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图注:上图(A)显示<br>了一名63岁的黑人男性患者的主要实验室检查结果,该患者因<br>与新型冠状病毒疾病(COVID-19)相关的急性呼吸窘迫而入院<br>。他迅速发展为急性肾损伤,无血流动力学损害。他的<br>呼吸状况得到改善,但炎症综合症持续存在,肾功能进一步<br>恶化。下图B,C和<br>D 显示了他的肾脏活检的说明性图像。光学显微镜研究(FAOG染色,原始放大倍数200倍(B和D)和400倍<br>(C))显示:1)严重的肾小球塌陷(局部节段性肾小球硬化),<br>其特征是毛细血管环(B和C)整体塌陷和<br>从肥厚,增生的足细胞<br>(“鹅卵石图案”,*,B)的基底膜上脱离,其中包含大量蛋白质重吸收液泡(*,<br>C)。2)急性肾小管病变(D),伴有局灶性肾小管坏死,扩张和存在<br>重度蛋​​白尿的肾小管内重吸收液泡(*,D)。<br>免疫荧光研究未显示任何明显的免疫沉淀。<br>电子显微镜研究(E,x 15000和F,x 73000)揭示了足细胞<br>胞质液泡中包含许多球形颗粒(*,E),尺寸在50至<br>110 nm 之间,并被9-10 nm(*,F)的尖峰包围。这些颗粒具有典型的<br>报告出现了新兴的严重急性<br>呼吸系统综合症冠状病毒2(SARS-CoV-2)3 病毒包涵体的出现(“日冕”)。<br>缩写:SCr,血清肌酐。上/下 尿蛋白超过肌酐的比例。UAlb / Cr,尿<br>白蛋白超过肌酐比率。CRP,C反应蛋白。SARS-CoV-2,严重急性呼吸系统<br>综合症冠状病毒2.白细胞,白细胞计数。PN,多核中性粒细胞。淋巴液,<br>淋巴细胞。血红蛋白,血红蛋白。Plt,血小板计数。IL,白介素。干扰素,干扰素。CCL,CXCL,TNF,<br>肿瘤坏死因子。CH50,溶血补体活性为50%。AP50,替代途径<br>活性为50%。sC5b-9,可溶性C5b-9。HIV,人类免疫缺陷病毒。CMV,巨细胞病毒。
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Legend to Figure:<br>Main laboratory results of a 63-year old black male patient admitted for acute respiratory<br>distress associated to novel coronavirus disease (COVID-19) are shown in the upper panel<br>(A). He rapidly developed acute kidney injury without hemodynamic compromise. His<br>respiratory status improved but inflammatory syndrome persisted and renal function further<br>deteriorated. Illustrative images of his kidney biopsy are shown in the lower panel B, C and<br>D). Light microscopy study (FAOG stain, original magnification x 200 (B and D) and x 400<br>(C)) showed: 1) a severe collapsing glomerulopathy (focal segmental glomerulosclerosis)<br>characterized by the global collapse of shrinking capillary loops (B and C) and the<br>detachment from the basement membrane of hypertrophic, proliferating podocytes<br>(“cobblestone pattern”, *, B) which contained numerous proteins reabsorption vacuoles (*,<br>C). 2) acute tubular lesions (D) with focal tubular necrosis, dilatation and the presence of<br>intratubular reabsorption vacuoles (*, D) reflecting the heavy proteinuria.<br>Immunofluorescence study did not show any significant immune deposits.<br>Electron microscopy study (E, x 15000 and F, x 73000) disclosed within the podocytes<br>cytoplasm vacuoles containing numerous spherical particles (*, E) measuring between 50 to<br>110 nm and surrounded by spikes measuring 9-10nm (*, F). These particles have the typical<br>appearance (“solar corona”) of viral inclusion bodies reported with the emerging severe acute<br>respiratory syndrome coronavirus 2 (SARS-CoV-2)3.<br>Abbreviations: SCr, serum creatinine. UP/Cr. Urinary protein over creatinine ratio. UAlb/Cr, urinary<br>albumin over creatinine ratio. CRP, C-reactive protein. SARS-CoV-2, severe acute respiratory<br>syndrome coronavirus 2. WBC, white blood cell count. PN, polynuclear neutrophils. Lym,<br>lymphocytes. Hb, haemoglobin. Plt, platelet count. IL, interleukin. IFN, interferon. CCL, CXCL, TNF,<br>tumor necrosis factor. CH50, haemolytic complement activity 50%. AP50, alternative pathway<br>activity 50%. sC5b-9, soluble C5b-9. HIV, human immunodeficiency virus. CMV, cytomegalovirus.
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结果 (简体中文) 3:[复制]
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图例到图:<br>63岁黑人男性急性呼吸道疾病患者主要实验室检查结果分析<br>与新型冠状病毒病(COVID-19)相关的危象如上图所示<br>(A) 是的。他迅速发展成急性肾损伤,血流动力学没有损害。伊斯<br>呼吸状况改善,但炎性综合征持续,肾功能进一步改善<br>恶化了。肾活检的说明性图像显示在下面板B、C和<br>D) 是的。光学显微镜研究(FAOG染色,原始放大倍率x 200(B和D)和x 400<br>(C) 显示:1)严重的塌陷性肾小球疾病(局灶节段性肾小球硬化)<br>以收缩的毛细血管环(B和C)的整体塌陷和<br>肥大增生足细胞基膜脱离<br>(“鹅卵石模式”,*,B)含有大量蛋白质重吸收泡(*,<br>C) 是的。2) 急性肾小管病变(D)伴局灶性肾小管坏死、扩张和<br>肾小管内再吸收泡(*,D)反映严重蛋白尿。<br>免疫荧光研究没有显示任何明显的免疫沉淀。<br>足细胞内的电子显微镜研究(E,x 15000和F,x 73000)<br>细胞质空泡,含有大量球形颗粒(*,E),大小在50到<br>110纳米,周围有9-10纳米的尖峰(*,F)。这些粒子具有典型的<br>病毒包涵体出现(“日冕”)并伴有严重急性期<br>呼吸综合征冠状病毒2(SARS-CoV-2)3。<br>缩写:SCr,血清肌酐。尿蛋白高于肌酐比值。尿白蛋白/肌酐<br>白蛋白高于肌酐比率。C反应蛋白。SARS-CoV-2,严重急性呼吸系统<br>冠状病毒2综合征。白细胞计数。PN,多核中性粒细胞。莱姆,<br>淋巴细胞。血红蛋白,血红蛋白。血小板计数。白细胞介素。干扰素。CCL,CXCL,肿瘤坏死因子,<br>肿瘤坏死因子。CH50,溶血性补体活性50%。AP50,替代途径<br>活动50%。sC5b-9,可溶性C5b-9。艾滋病毒,人体免疫缺陷病毒。巨细胞病毒。<br>
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