Other mechanisms of resistance conveying the CRE phenotype identified for K. pneumoniae were the metallo-β-lactamase enzyme and the OXA-like β-lactamase enzyme.See Section 11.2 for further details on the mechanism of carbapenem resistance for other CRE pathogens identified in this study.See Post-text Table 2.6.1 for a summary of the mechanism for carbapenem resistance for the CRE qualifying pathogen for All Cases.cUTI/AP(N=76)Patient-specific factorsPrior culture positive for CREDuration of hospitalization - mean(SD)[2]Presence of neutropenia[4]Diabetes with end organ damageRequirement for dialysisThe sources of bacteremia were IV catheter(47/140[33.6%]); unknown(23/140[16.4%); cUTI/AP(12/140[8.6%]); HABP(5/140[3.6%]); VABP(5/140[3.6%]); and other(48/140[34.3%]).Duration of hospitalization prior to index CRE infection.Immunocompromised condition included hematologic malignancy, prior bone marrow transplant, or received immunosuppressive therapy, such as cancer chemotherapy, anti-rejection medications for transplant