据认为NSCLC患者体内血管内皮生长因子（VEGF）水平过高，使其与血

It is believed that the level of vascular endothelial growth factor (VEGF) in patients with NSCLC is too high, causing it to bind to vascular endothelial cell growth factor receptor 2 (VEGFR-2), resulting in an increase in the number of tumor new blood vessels, increased vascular permeability, and increased endothelial cells Vitality promotes the chemotaxis and metastasis of tumor cells through complex regulatory mechanisms.

It is believed that NSCLC patients have too high level of vascular endothorkin growth factor (VEGF), which binds it with vascular endothortic cell growth factor subject 2 (VEGFR-2), resulting in an increase in the number of tumor new blood vessels, increased vascular permeability, enhance endoskin cell vitality, promote the trend of tumor cells, metastasis through complex regulatory mechanisms.

It is believed that the level of vascular endothelial growth factor (VEGF) in NSCLC patients is too high, which makes it combine with vascular endothelial growth factor receptor 2 (VEGFR-2), resulting in an increase in the number of tumor neovascularization, vascular permeability, enhance the vitality of endothelial cells, promote the chemotaxis and metastasis of tumor cells through complex regulatory mechanisms.<br>